Combined Deletion of Macrophage ABCA1 and ABCG1 Leads to Massive Lipid Accumulation in Tissue Macrophages and Distinct Atherosclerosis at Relatively Low Plasma Cholesterol Levels-Reference-Cited by-同舟云学术

Combined Deletion of Macrophage ABCA1 and ABCG1 Leads to Massive Lipid Accumulation in Tissue Macrophages and Distinct Atherosclerosis at Relatively Low Plasma Cholesterol Levels

Published:2008-02 Issue:2 Volume:28 Page:258-264
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Out Ruud¹,Hoekstra Menno¹,Habets Kim¹,Meurs Illiana¹,de Waard Vivian¹,Hildebrand Reeni B.¹,Wang Yanan¹,Chimini Giovanna¹,Kuiper Johan¹,Van Berkel Theo J.C.¹,Van Eck Miranda¹

Affiliation:

1. From the Division of Biopharmaceutics (R.O., M.H., K.H., I.M., V.d.W., R.B.H., Y.W., J.K., T.J.C.V.B., and M.V.E.), Leiden/Amsterdam Center for Drug Research, Gorlaeus Laboratories, Leiden University, The Netherlands; and the Centre d’Immunologie de Marseille Luminy (G.C.), Institut National de la Santé et de la Recherche Médicale, Centre National de la Recherche Scientifique, Université de la Méditerranée, Marseille, France.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference32 articles.

1. The plasma lecithin:cholesterol acyltransferase reaction

2. ABC transporters in cellular lipid trafficking

3. Scavenger receptor BI and ATP-binding cassette transporter A1 in reverse cholesterol transport and atherosclerosis

4. ABCA1 Is the cAMP-inducible Apolipoprotein Receptor That Mediates Cholesterol Secretion from Macrophages

5. Increased Atherosclerosis in Hyperlipidemic Mice With Inactivation of ABCA1 in Macrophages

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