The Intrinsic Complement Regulator Decay-Accelerating Factor Modulates the Biological Response to Vascular Injury-Reference-Cited by-同舟云学术

The Intrinsic Complement Regulator Decay-Accelerating Factor Modulates the Biological Response to Vascular Injury

Published:2010-06 Issue:6 Volume:30 Page:1196-1202
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Sakuma Masashi¹,Morooka Toshifumi¹,Wang Yunmei¹,Shi Can¹,Croce Kevin¹,Gao Huiyun¹,Strainic Michael¹,Medof M. Edward¹,Simon Daniel I.¹

Affiliation:

1. From the University Hospitals Harrington-McLaughlin Heart and Vascular Institute and Case Cardiovascular Center (M.S., T.M., Y.W., C.S., H.G., and D.I.S.), Case Western Reserve University School of Medicine, Cleveland, Ohio; the Division of Cardiovascular Medicine (K.C.), Brigham and Women’s Hospital, Harvard Medical School, Boston, Mass; and the Institute of Pathology (M.S. and M.E.M.), Case Western Reserve University School of Medicine, Cleveland, Ohio.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference44 articles.

1. A Critical Role of Platelet Adhesion in the Initiation of Atherosclerotic Lesion Formation

2. Decreased neointimal formation in Mac-1–/– mice reveals a role for inflammation in vascular repair after angioplasty

3. Targeting Platelet–Leukocyte Interactions

4. Leukocyte Engagement of Platelet Glycoprotein Ibα via the Integrin Mac-1 Is Critical for the Biological Response to Vascular Injury

5. Evolution of the lectin–complement pathway and its role in innate immunity

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