Long-Term Regulation of ENaC Expression in Kidney by Angiotensin II-Reference-Cited by-同舟云学术

Long-Term Regulation of ENaC Expression in Kidney by Angiotensin II

Published:2003-05 Issue:5 Volume:41 Page:1143-1150
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Beutler Kathleen T.¹,Masilamani Shyama¹,Turban Sharon¹,Nielsen Jakob¹,Brooks Heddwen L.¹,Ageloff Shana¹,Fenton Robert A.¹,Packer Randall K.¹,Knepper Mark A.¹

Affiliation:

1. From the Laboratory of Kidney and Electrolyte Metabolism, NHLBI, National Institutes of Health (K.T.B., S.M., S.T., J.N., H.L.B., S.A., R.A.F., M.A.K.), Bethesda, Md; and Department of Biological Sciences, George Washington University (K.T.B., R.K.P.), Washington, DC.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference40 articles.

1. Dose-dependent stimulation and inhibition of proximal tubular sodium reabsorption by angiotensin II in the rat kidney

2. Angiotensin stimulates glucose and fluid absorption by rat proximal straight tubules.

3. Angiotensin II stimulates both Na(+)-H+ exchange and Na+/HCO3- cotransport in the rabbit proximal tubule.

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