Nebivolol Inhibits Superoxide Formation by NADPH Oxidase and Endothelial Dysfunction in Angiotensin II

Nebivolol Inhibits Superoxide Formation by NADPH Oxidase and Endothelial Dysfunction in Angiotensin II–Treated Rats

Published:2006-10 Issue:4 Volume:48 Page:677-684
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Oelze Matthias¹,Daiber Andreas¹,Brandes Ralf P.¹,Hortmann Marcus¹,Wenzel Philip¹,Hink Ulrich¹,Schulz Eberhard¹,Mollnau Hanke¹,von Sandersleben Alexandra¹,Kleschyov Andrei L.¹,Mülsch Alexander¹,Li Huige¹,Förstermann Ulrich¹,Münzel Thomas¹

Affiliation:

1. From the Johannes Gutenberg University Hospital (M.O., A.D., P.W., U.H., E.S., H.M., A.L.K., A.M., T.M.), Division of Cardiology, Mainz, Germany; J.W. Goethe-University (R.P.B.), Institut für Kardiovaskuläre Physiologie, Frankfurt am Main, Germany; Institut für Pharmakologie (M.H., H.L., U.F.), Johannes Gutenberg-Universität Mainz, Mainz, Germany; and University Hospital Eppendorf (A.v.S.), Division of Cardiology, Hamburg, Germany.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference28 articles.

1. Nitric oxide, oxidative stress and hypertension: a complex equation

2. Reactive oxygen species in vascular biology: role in arterial hypertension

3. Angiotensin II-mediated hypertension in the rat increases vascular superoxide production via membrane NADH/NADPH oxidase activation. Contribution to alterations of vasomotor tone.

4. Effects of Angiotensin II Infusion on the Expression and Function of NAD(P)H Oxidase and Components of Nitric Oxide/cGMP Signaling

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