Reduced NOS3 Phosphorylation Mediates Reduced NO/cGMP Signaling in Mesenteric Arteries of Deoxycorticosterone Acetate-Salt Hypertensive Rats-Reference-Cited by-同舟云学术

Reduced NOS3 Phosphorylation Mediates Reduced NO/cGMP Signaling in Mesenteric Arteries of Deoxycorticosterone Acetate-Salt Hypertensive Rats

Published:2004-05 Issue:5 Volume:43 Page:1080-1085
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Sasser Jennifer M.¹,Sullivan Jennifer C.¹,Elmarakby Ahmed A.¹,Kemp Bruce E.¹,Pollock David M.¹,Pollock Jennifer S.¹

Affiliation:

1. From the Department of Pharmacology (J.M.S., J.C.S.,A.A.E.,D.M.P., J.S.P.), Vascular Biology Center (J.C.S., D.M.P., J.S.P.) and Department of Surgery (D.M.P.), Medical College of Georgia, Augusta; The St. Vincent’s Institute of Medical Research (B.E.K.) Fitzroy, Australia.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference31 articles.

1. Nitric oxide synthase generates superoxide and nitric oxide in arginine-depleted cells leading to peroxynitrite-mediated cellular injury.

2. Oxidation of tetrahydrobiopterin leads to uncoupling of endothelial cell nitric oxide synthase in hypertension

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