Aberrant Mitochondrial Fission Is Maladaptive in Desmin Mutation–Induced Cardiac Proteotoxicity-Reference-Cited by-同舟云学术

Aberrant Mitochondrial Fission Is Maladaptive in Desmin Mutation–Induced Cardiac Proteotoxicity

Published:2018-07-17 Issue:14 Volume:7 Page:
ISSN:2047-9980
Container-title:Journal of the American Heart Association
language:en
Short-container-title:J Am Heart Assoc

Author:

Alam Shafiul¹,Abdullah Chowdhury S.¹,Aishwarya Richa²,Miriyala Sumitra³,Panchatcharam Manikandan³,Peretik Jonette M.¹,Orr A. Wayne¹²³,James Jeanne⁴,Robbins Jeffrey⁵,Bhuiyan Md. Shenuarin¹²

Affiliation:

1. Department of Pathology and Translational Pathobiology Louisiana State University Health Sciences Center Shreveport LA

2. Department of Molecular and Cellular Physiology Louisiana State University Health Sciences Center Shreveport LA

3. Department of Cellular Biology and Anatomy Louisiana State University Health Sciences Center Shreveport LA

4. Division of Pediatric Cardiology Medical College of Wisconsin Milwaukee WI

5. Division of Molecular Cardiovascular Biology Cincinnati Children's Hospital Cincinnati OH

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference59 articles.

1. Association of Mitochondria with Plectin and Desmin Intermediate Filaments in Striated Muscle

2. A network of transverse and longitudinal intermediate filaments is associated with sarcomeres of adult vertebrate skeletal muscle.

3. The biology of desmin filaments: how do mutations affect their structure, assembly, and organisation?

4. Cardiovascular Lesions and Skeletal Myopathy in Mice Lacking Desmin

5. Disruption of muscle architecture and myocardial degeneration in mice lacking desmin.

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