Cardiac Fibrosis in Proteotoxic Cardiac Disease is Dependent Upon Myofibroblast TGF‐β Signaling-Reference-Cited by-同舟云学术

Cardiac Fibrosis in Proteotoxic Cardiac Disease is Dependent Upon Myofibroblast TGF‐β Signaling

Published:2018-10-16 Issue:20 Volume:7 Page:
ISSN:2047-9980
Container-title:Journal of the American Heart Association
language:en
Short-container-title:J Am Heart Assoc

Author:

Bhandary Bidur¹,Meng Qinghang¹,James Jeanne²,Osinska Hanna¹,Gulick James¹,Valiente‐Alandi Iñigo¹,Sargent Michelle A.¹,Bhuiyan Md. Shenuarin³,Blaxall Burns C.¹,Molkentin Jeffery D.¹,Robbins Jeffrey¹

Affiliation:

1. Division of Molecular Cardiovascular Biology Cincinnati Children's Hospital Cincinnati OH

2. Division of Pediatric Cardiology Medical College of Wisconsin Milwaukee WI

3. Department of Pathology and Translational Pathobiology Louisiana State University Health Sciences Center Shreveport LA

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference35 articles.

1. Global, regional, and national life expectancy, all-cause mortality, and cause-specific mortality for 249 causes of death, 1980–2015: a systematic analysis for the Global Burden of Disease Study 2015

2. Novel therapeutic strategies targeting fibroblasts and fibrosis in heart disease

3. Myofibroblast-mediated mechanisms of pathological remodelling of the heart

4. Dynamics of Cell Generation and Turnover in the Human Heart

5. Revisiting Cardiac Cellular Composition

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