Role of Osteopontin in Cardiac Fibrosis and Remodeling in Angiotensin II-Induced Cardiac Hypertrophy-Reference-Cited by-同舟云学术

Role of Osteopontin in Cardiac Fibrosis and Remodeling in Angiotensin II-Induced Cardiac Hypertrophy

Published:2004-06 Issue:6 Volume:43 Page:1195-1201
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Matsui Yutaka¹,Jia Nan¹,Okamoto Hiroshi¹,Kon Shigeyuki¹,Onozuka Hisao¹,Akino Masatoshi¹,Liu Lizhi¹,Morimoto Junko¹,Rittling Susan R.¹,Denhardt David¹,Kitabatake Akira¹,Uede Toshimitsu¹

Affiliation:

1. From the Department of Cardiovascular Medicine (Y.M., N.J., H. Okamoto, H. Onozuka, M.A., L.L., A.K.), Hokkaido University Graduate School of Medicine, Sapporo, Japan; Division of Molecular Immunology (Y.M., N.J., S.K., J.M., T.U.), Institute for Genetic Medicine, Hokkaido University, Sapporo, Japan; Departments of Genetics (S.R.R.) and Cell Biology and Neuroscience (D.D.), Rutgers University, Piscataway, NJ.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference20 articles.

1. Osteopontin is produced by rat cardiac fibroblasts and mediates A(II)-induced DNA synthesis and collagen gel contraction.

2. Cross-linking of Osteopontin by Tissue Transglutaminase Increases Its Collagen Binding Properties

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