Cardiac Hypertrophy Is Associated With Decreased eNOS Expression in Angiotensin AT 2 Receptor

Cardiac Hypertrophy Is Associated With Decreased eNOS Expression in Angiotensin AT 2 Receptor–Deficient Mice

Published:2003-12 Issue:6 Volume:42 Page:1177-1182
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Brede Marc¹,Roell Wilhelm¹,Ritter Oliver¹,Wiesmann Frank¹,Jahns Roland¹,Haase Axel¹,Fleischmann Bernd K.¹,Hein Lutz¹

Affiliation:

1. From the Institut für Pharmakologie und Toxikologie (M.B., R.J., L.H.), Medizinische Klinik (O.R., F.W.), Medizinische Poliklinik (R.J.), and Physikalisches Institut (F.W., A.H.), Universität Würzburg, Würzburg; the Klinik und Poliklinik für Herzchirurgie (W.R.), Universität Bonn, Bonn; and Institut für Neurophysiologie (B.K.F.), Universität Köln, Köln, Germany.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference34 articles.

1. The angiotensin II type 2 (AT2) receptor antagonizes the growth effects of the AT1 receptor: gain-of-function study using gene transfer.

2. The angiotensin AT2-receptor mediates inhibition of cell proliferation in coronary endothelial cells.

3. T cell–mediated Fas-induced keratinocyte apoptosis plays a key pathogenetic role in eczematous dermatitis

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