Follistatin-Like 1 in Chronic Systolic Heart Failure-Reference-Cited by-同舟云学术

Follistatin-Like 1 in Chronic Systolic Heart Failure

Published:2011-09 Issue:5 Volume:4 Page:621-627
ISSN:1941-3289
Container-title:Circulation: Heart Failure
language:en
Short-container-title:Circ Heart Fail

Author:

El-Armouche Ali¹,Ouchi Noriyuki¹,Tanaka Komei¹,Doros Gheorghe¹,Wittköpper Katrin¹,Schulze Thomas¹,Eschenhagen Thomas¹,Walsh Kenneth¹,Sam Flora¹

Affiliation:

1. From the Department of Pharmacology, University Medical Center Göttingen, Göttingen, Germany (A.E.-A., K.W.); Whitaker Cardiovascular Institute (N.O., K.T., K.W., F.S.) and Cardiovascular Section and Evans Department of Medicine (F.S.), Boston University School of Medicine, and Biostatistics Department (G.D.), Boston University School of Public Health, Boston, MA; and Department of Experimental and Clinical Pharmacology (T.S., T.E.), University Medical Center Hamburg-Eppendorf, Hamburg, Germany.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference19 articles.

1. Cloning from a mouse osteoblastic cell line of a set of transforming-growth-factor-beta1-regulated genes, one of which seems to encode a follistatin-related polypeptide

2. DIP2A Functions as a FSTL1 Receptor

3. Expression of a TGF-β1 inducible gene, TSC-36, causes growth inhibition in human lung cancer cell lines

4. Ameliorative effects of follistatin-related protein/TSC-36/FSTL1 on joint inflammation in a mouse model of arthritis

5. An Immunomodulatory Role for Follistatin-Like 1 in Heart Allograft Transplantation

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