Sodium Channel Remodeling in Subcellular Microdomains of Murine Failing Cardiomyocytes-Reference-Cited by-同舟云学术

Sodium Channel Remodeling in Subcellular Microdomains of Murine Failing Cardiomyocytes

Published:2017-12-02 Issue:12 Volume:6 Page:
ISSN:2047-9980
Container-title:Journal of the American Heart Association
language:en
Short-container-title:JAHA

Author:

Rivaud Mathilde R.¹²³,Agullo‐Pascual Esperanza²,Lin Xianming²,Leo‐Macias Alejandra²,Zhang Mingliang²,Rothenberg Eli⁴,Bezzina Connie R.¹,Delmar Mario²,Remme Carol Ann¹

Affiliation:

1. Heart Center, Department of Clinical and Experimental Cardiology, Academic Medical Center, University of Amsterdam, the Netherlands

2. Division of Cardiology, New York University Medical Center, New York, NY

3. Department of Medical Physiology, University Medical Center Utrecht, Utrecht, the Netherlands

4. Department of Biochemistry and Molecular Pharmacology, NYU‐School of Medicine, New York, NY

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference50 articles.

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3. Increased late sodium current in myocytes from a canine heart failure model and from failing human heart

4. Increased late sodium currents are related to transcription of neuronal isoforms in a pressure-overload model

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