Recombinant Human ADAMTS13 Treatment Improves Myocardial Remodeling and Functionality After Pressure Overload Injury in Mice

Author:

Witsch Thilo123,Martinod Kimberly124,Sorvillo Nicoletta12,Portier Irina4,De Meyer Simon F.4,Wagner Denisa D.125

Affiliation:

1. Program in Cellular and Molecular Medicine, Boston Children's Hospital, Boston, MA

2. Department of Pediatrics, Harvard Medical School, Boston, MA

3. Department of Cardiology and Angiology I, Heart Center, University of Freiburg, Freiburg, Germany

4. Laboratory for Thrombosis Research, KU Leuven Campus Kulak Kortrijk, Kortrijk, Belgium

5. Division of Hematology/Oncology, Boston Children's Hospital, Boston, MA

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference44 articles.

1. Epidemiology and risk profile of heart failure

2. Myocardial ischemia-reperfusion injury: a neglected therapeutic target

3. Acute inflammatory reaction after myocardial ischemic injury and reperfusion: development and use of a neutrophil‐specific antibody;Hawkins HK;Am J Pathol,1996

4. Ly6Clow and Not Ly6Chigh Macrophages Accumulate First in the Heart in a Model of Murine Pressure-Overload

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