Collecting Duct Nitric Oxide Synthase 1ß Activation Maintains Sodium Homeostasis During High Sodium Intake Through Suppression of Aldosterone and Renal Angiotensin II Pathways-Reference-Cited by-同舟云学术

Collecting Duct Nitric Oxide Synthase 1ß Activation Maintains Sodium Homeostasis During High Sodium Intake Through Suppression of Aldosterone and Renal Angiotensin II Pathways

Published:2017-10-11 Issue:10 Volume:6 Page:
ISSN:2047-9980
Container-title:Journal of the American Heart Association
language:en
Short-container-title:JAHA

Author:

Hyndman Kelly A.¹,Mironova Elena V.²,Giani Jorge F.³,Dugas Courtney¹,Collins Jessika¹,McDonough Alicia A.⁴,Stockand James D.²,Pollock Jennifer S.¹

Affiliation:

1. Cardio‐Renal Physiology and Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, AL

2. Department of Cellular and Integrative Physiology, University of Texas Health Science Center at San Antonio, TX

3. Department of Biomedical Sciences, Cedars‐Sinai Medical Center, Los Angeles, CA

4. Department of Cell and Neurobiology, Keck School of Medicine, University of Southern California, Los Angeles, CA

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference44 articles.

1. Renal Collecting Duct NOS1 Maintains Fluid–Electrolyte Homeostasis and Blood Pressure

2. Splice variants of neuronal nitric oxide synthase are present in the rat kidney

3. Distinct regulation of inner medullary collecting duct nitric oxide production from mice and rats

4. Dynamin-2 is a novel NOS1β interacting protein and negative regulator in the collecting duct

5. NOS1-dependent negative feedback regulation of the epithelial sodium channel in the collecting duct

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