Experimentally Increasing Titin Compliance in a Novel Mouse Model Attenuates the Frank-Starling Mechanism But Has a Beneficial Effect on Diastole-Reference-Cited by-同舟云学术

Experimentally Increasing Titin Compliance in a Novel Mouse Model Attenuates the Frank-Starling Mechanism But Has a Beneficial Effect on Diastole

Published:2014-05-13 Issue:19 Volume:129 Page:1924-1936
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Methawasin Mei¹,Hutchinson Kirk R.¹,Lee Eun-Jeong¹,Smith John E.¹,Saripalli Chandra¹,Hidalgo Carlos G.¹,Ottenheijm Coen A.C.¹,Granzier Henk¹

Affiliation:

1. From the Sarver Molecular Cardiovascular Research Program, Departments of Physiology and Cellular and Molecular Medicine, University of Arizona, Tucson, AZ (M.M., K.R.H., E.-J.L., J.E.S., C.S., C.G.H., C.A.C.O., H.G.); and Department of Physiology, VU University Medical Center, Amsterdam, the Netherlands (C.A.C.O.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference37 articles.

1. Cardiac Titin

2. RBM20, a gene for hereditary cardiomyopathy, regulates titin splicing

3. Developmental Control of Titin Isoform Expression and Passive Stiffness in Fetal and Neonatal Myocardium

4. Developmentally Regulated Switching of Titin Size Alters Myofibrillar Stiffness in the Perinatal Heart

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