Type 5 Phosphodiesterase Inhibition-Reference-Cited by-同舟云学术

Type 5 Phosphodiesterase Inhibition

Published:2005-10-25 Issue:17 Volume:112 Page:2589-2591
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Semigran Marc J.¹

Affiliation:

1. From the Cardiology Division, Department of Medicine, Massachusetts General Hospital, Harvard Medical School, Boston, Mass.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Link

https://www.ahajournals.org/doi/pdf/10.1161/CIRCULATIONAHA.105.577627

Reference22 articles.

1. The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine

2. Human prepro atrial natriuretic factors 26–55, 56–92, and 104–123 increase renal guanylate cyclase activity

3. Sildenafil Inhibits β-Adrenergic–Stimulated Cardiac Contractility in Humans

4. Mery PF, Pavoine C, Belhassen L, Pecker F, Fischmeister R. Nitric oxide regulates cardiac Ca2+ current: involvement of cGMP-inhibited and cGMP-stimulated phosphodiesterases through guanylate cyclase activation. J Biol Chem. 1994; 268: 26286–26295.

5. Pertussis toxin-sensitive G proteins influence nitric oxide synthase III activity and protein levels in rat heart.

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