Mitochondrial Transporter ATP Binding Cassette Mitochondrial Erythroid Is a Novel Gene Required for Cardiac Recovery After Ischemia/Reperfusion-Reference-Cited by-同舟云学术

Mitochondrial Transporter ATP Binding Cassette Mitochondrial Erythroid Is a Novel Gene Required for Cardiac Recovery After Ischemia/Reperfusion

Published:2011-08-16 Issue:7 Volume:124 Page:806-813
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Liesa Marc¹,Luptak Ivan¹,Qin Fuzhong¹,Hyde Brigham B.¹,Sahin Ergun¹,Siwik Deborah A.¹,Zhu Zhengkun¹,Pimentel David R.¹,Xu X. Julia¹,Ruderman Neil B.¹,Huffman Karl D.¹,Doctrow Susan R.¹,Richey Lauren¹,Colucci Wilson S.¹,Shirihai Orian S.¹

Affiliation:

1. From the Department of Medicine, Obesity and Diabetes Research Centers (M.L., B.B.H., X.J.X., N.B.R., O.S.S.), Cardiovascular Section (I.L., F.Q., D.A.S., Z.Z., D.R.P., W.S.C.), and Pulmonary Center (K.D.H., S.R.D.), Mitochondria ARC, Boston University School of Medicine; Department of Medical Oncology, Dana-Farber Cancer Institute, Harvard Medical School (E.S.); and Division of Laboratory Animal Medicine, Tufts University (L.R), Boston, MA.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference37 articles.

1. Targeting an antioxidant to mitochondria decreases cardiac ischemia‐reperfusion injury

2. Mitochondrial Complex II in the Post-ischemic Heart

3. Mitochondria as a Target for the Cardioprotective Effects of Nitric Oxide in Ischemia–Reperfusion Injury

4. Cardioprotection by metabolic shut-down and gradual wake-up

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