Plasmin Cleavage of von Willebrand Factor as an Emergency Bypass for ADAMTS13 Deficiency in Thrombotic Microangiopathy-Reference-Cited by-同舟云学术

Plasmin Cleavage of von Willebrand Factor as an Emergency Bypass for ADAMTS13 Deficiency in Thrombotic Microangiopathy

Published:2014-03-25 Issue:12 Volume:129 Page:1320-1331
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Tersteeg Claudia¹,de Maat Steven¹,De Meyer Simon F.¹,Smeets Michel W.J.¹,Barendrecht Arjan D.¹,Roest Mark¹,Pasterkamp Gerard¹,Fijnheer Rob¹,Vanhoorelbeke Karen¹,de Groot Philip. G.¹,Maas Coen¹

Affiliation:

1. From the Laboratory of Clinical Chemistry and Haematology (C.T., S.d.M., M.W.J.S., A.D.B., M.R., R.F., P.G.d.G., C.M.) and Laboratory of Experimental Cardiology (C.T., G.P.), UMC Utrecht, Utrecht, The Netherlands; and Laboratory for Thrombosis Research, IRF Life Sciences, KU Leuven Kulak, Kortrijk, Belgium (S.F.D.M., K.V.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference26 articles.

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2. von Willebrand factor: a reduced and alkylated 52/48-kDa fragment beginning at amino acid residue 449 contains the domain interacting with platelet glycoprotein Ib.;Fujimura Y;J Biol Chem,1986

3. Partial purification and characterization of a protease from human plasma cleaving von Willebrand factor to fragments produced by in vivo proteolysis.;Furlan M;Blood,1996

4. Platelet-VWF complexes are preferred substrates of ADAMTS13 under fluid shear stress

5. von Willebrand Factor–Cleaving Protease in Thrombotic Thrombocytopenic Purpura and the Hemolytic–Uremic Syndrome

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