Effect of the Proprotein Convertase Subtilisin/Kexin 9 Monoclonal Antibody, AMG 145, in Homozygous Familial Hypercholesterolemia-Reference-Cited by-同舟云学术

Effect of the Proprotein Convertase Subtilisin/Kexin 9 Monoclonal Antibody, AMG 145, in Homozygous Familial Hypercholesterolemia

Published:2013-11-05 Issue:19 Volume:128 Page:2113-2120
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Stein Evan A.¹,Honarpour Narimon¹,Wasserman Scott M.¹,Xu Feng¹,Scott Rob¹,Raal Frederick J.¹

Affiliation:

1. From the Metabolic and Atherosclerosis Research Center, Cincinnati, OH (E.A.S.); Global Development (N.H., S.M.W., R.S.) and Biostatistics (F.X.), Amgen Inc, Thousand Oaks, CA; and Carbohydrate & Lipid Metabolism Research Unit, Faculty of Health Sciences, University of Witwatersrand, Johannesburg, South Africa (F.J.R.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference31 articles.

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2. Mutations in PCSK9 cause autosomal dominant hypercholesterolemia

3. Monogenic hypercholesterolemia: new insights in pathogenesis and treatment

4. Low-Density Lipoprotein Receptor Gene Familial Hypercholesterolemia Variant Database: Update and Pathological Assessment

5. A dose-titration and comparative study of rosuvastatin and atorvastatin in patients with homozygous familial hypercholesterolaemia

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