Critical Role of Bone Marrow Apoptosis-Associated Speck-Like Protein, an Inflammasome Adaptor Molecule, in Neointimal Formation After Vascular Injury in Mice-Reference-Cited by-同舟云学术

Critical Role of Bone Marrow Apoptosis-Associated Speck-Like Protein, an Inflammasome Adaptor Molecule, in Neointimal Formation After Vascular Injury in Mice

Published:2008-06-17 Issue:24 Volume:117 Page:3079-3087
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Yajima Noriyuki¹,Takahashi Masafumi¹,Morimoto Hajime¹,Shiba Yuji¹,Takahashi Yasuko¹,Masumoto Junya¹,Ise Hirohiko¹,Sagara Junji¹,Nakayama Jun¹,Taniguchi Shun'ichiro¹,Ikeda Uichi¹

Affiliation:

1. From the Departments of Cardiovascular Medicine (N.Y., M.T., H.M., Y.S., H.I., U.I.) and Molecular Oncology (Y.T., J.S., S.T.), Shinshu University Graduate School of Medicine, and Department of Pathology (J.M., J.N.), Shinshu University School of Medicine, Matsumoto, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference34 articles.

1. Interleukin 1 and interleukin 18 as mediators of inflammation and the aging process

2. Deficiency of Interleukin-1 Receptor Antagonist Promotes Neointimal Formation After Injury

3. Neutralization of Interleukin-18 Inhibits Neointimal Formation in a Rat Model of Vascular Injury

4. The Inflammasome

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