Downregulation of Apoptosis-Inducing Factor in Harlequin Mutant Mice Sensitizes the Myocardium to Oxidative Stress–Related Cell Death and Pressure Overload–Induced Decompensation-Reference-Cited by-同舟云学术

Downregulation of Apoptosis-Inducing Factor in Harlequin Mutant Mice Sensitizes the Myocardium to Oxidative Stress–Related Cell Death and Pressure Overload–Induced Decompensation

Published:2005-06-24 Issue:12 Volume:96 Page:
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

van Empel Vanessa P.M.¹,Bertrand Anne T.¹,van der Nagel Roel¹,Kostin Sawa¹,Doevendans Pieter A.¹,Crijns Harry J.¹,de Wit Elly¹,Sluiter Wim¹,Ackerman Susan L.¹,De Windt Leon J.¹

Affiliation:

1. From the Hubrecht Laboratory and Interuniversity Cardiology Institute Netherlands (V.P.M.v.E., A.T.B., R.v.d.N., L.J.D.W.), Royal Netherlands Academy of Arts and Sciences, Utrecht, the Netherlands; Max-Planck-Institute (S.K.), Bad Nauheim, Germany; Heart Lung Center Utrecht (P.A.D.), the Netherlands; Department of Cardiology (H.J.C.), University Hospital Maastricht, the Netherlands; Department of Biochemistry (E.d.W., W.S.), Erasmus MC, Rotterdam, the Netherlands; and The Jackson Laboratory (S.L.A.)...

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference40 articles.

1. A mechanistic role for cardiac myocyte apoptosis in heart failure

2. Mitochondrial control of nuclear apoptosis.

3. Role of cytochrome c and dATP/ATP hydrolysis in Apaf-1-mediated caspase-9 activation and apoptosis

4. Apoptosis in heart failure: Release of cytochrome c from mitochondria and activation of caspase-3 in human cardiomyopathy

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