Gene Transfer of Neuronal Nitric Oxide Synthase to Carotid Body Reverses Enhanced Chemoreceptor Function in Heart Failure Rabbits-Reference-Cited by-同舟云学术

Gene Transfer of Neuronal Nitric Oxide Synthase to Carotid Body Reverses Enhanced Chemoreceptor Function in Heart Failure Rabbits

Published:2005-08-05 Issue:3 Volume:97 Page:260-267
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Li Yu-Long¹,Li Yi-Fan¹,Liu Dongmei¹,Cornish Kurtis G.¹,Patel Kaushik P.¹,Zucker Irving H.¹,Channon Keith M.¹,Schultz Harold D.¹

Affiliation:

1. From the Department of Cellular and Integrative Physiology (Y.-L.L., D.L., K.G.C., K.P.P., I.H.Z., H.D.S.), University of Nebraska Medical Center, Omaha; Division of Basic Biomedical Sciences (Y.-F.L.), University of South Dakota School of Medicine, Vermillion; and the Department of Cardiovascular Medicine (K.M.C.), University of Oxford, John Radcliffe Hospital, UK.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference47 articles.

1. Nitric oxide synthase and neuronal NADPH diaphorase are identical in brain and peripheral tissues.

2. Nitric oxide synthase isoforms and peripheral chemoreceptor stimulation in conscious rats

3. Nitric oxide and ventilatory response to hypoxia

4. Central and primary visceral afferents to nucleus tractus solitarii may generate nitric oxide as a membrane-permeant neuronal messenger

5. Localization and actions of nitric oxide in the cat carotid body

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