Hypoxia, BNip3 Proteins, and the Mitochondrial Death Pathway in Cardiomyocytes
Author:
Affiliation:
1. From the Department of Medicine, Johns Hopkins University, Baltimore, Md.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Link
https://www.ahajournals.org/doi/pdf/10.1161/01.RES.0000030195.38795.CF
Reference19 articles.
1. Inducible Expression of BNIP3 Provokes Mitochondrial Defects and Hypoxia-Mediated Cell Death of Ventricular Myocytes
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