Elevated Homocysteine Reduces Apolipoprotein A-I Expression in Hyperhomocysteinemic Mice and in Males With Coronary Artery Disease-Reference-Cited by-同舟云学术

Elevated Homocysteine Reduces Apolipoprotein A-I Expression in Hyperhomocysteinemic Mice and in Males With Coronary Artery Disease

Published:2006-03-03 Issue:4 Volume:98 Page:564-571
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Mikael Leonie G.¹,Genest Jacques¹,Rozen Rima¹

Affiliation:

1. From the Departments of Human Genetics and Pediatrics (L.G.M., R.R.), Montreal Children’s Hospital; and The Department of Medicine (J.G.), Royal Victoria Hospital, McGill University Health Center, Montreal, Canada.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference40 articles.

1. Effect of potentially modifiable risk factors associated with myocardial infarction in 52 countries (the INTERHEART study): case-control study

2. Homocysteine and Cardiovascular Disease

3. A candidate genetic risk factor for vascular disease: a common mutation in methylenetetrahydrofolate reductase

4. Mice deficient in methylenetetrahydrofolate reductase exhibit hyperhomocysteinemia and decreased methylation capacity, with neuropathology and aortic lipid deposition

5. Mice Deficient in Methylenetetrahydrofolate Reductase Exhibit Tissue-Specific Distribution of Folates

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