Tumor Necrosis Factor–Related Apoptosis-Inducing Ligand (TRAIL) Sequentially Upregulates Nitric Oxide and Prostanoid Production in Primary Human Endothelial Cells-Reference-Cited by-同舟云学术

Tumor Necrosis Factor–Related Apoptosis-Inducing Ligand (TRAIL) Sequentially Upregulates Nitric Oxide and Prostanoid Production in Primary Human Endothelial Cells

Published:2003-04-18 Issue:7 Volume:92 Page:732-740
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Zauli Giorgio¹,Pandolfi Assunta¹,Gonelli Arianna¹,Di Pietro Roberta¹,Guarnieri Simone¹,Ciabattoni Giovanni¹,Rana Rosalba¹,Vitale Marco¹,Secchiero Paola¹

Affiliation:

1. From the Department of Normal Human Morphology (G.Z., M.V.), University of Trieste, Trieste, Italy; the Department of Biomorphology (A.P., R.D.P., R.R.), “G. D’Annunzio” University of Chieti, Chieti Scalo, Chieti, Italy; the Department of Morphology and Embryology (A.G., P.S.), Human Anatomy Section, University of Ferrara, Ferrara, Italy; and the Department of Drug Sciences (G.C.), Laboratory of Cell Physiology (S.G.), “G. D’Annunzio” University of Chieti, Chieti, Italy.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference38 articles.

1. Differential regulation of TRAIL and CD95 ligand in transformed cells of the T and B lymphocyte lineage

2. Tumoricidal activity of tumor necrosis factor–related apoptosis–inducing ligand in vivo

3. Safety and antitumor activity of recombinant soluble Apo2 ligand

4. Control of TRAIL-Induced Apoptosis by a Family of Signaling and Decoy Receptors

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