Cardiac Sodium Channel Na v 1.5 Is Regulated by a Multiprotein Complex Composed of Syntrophins and Dystrophin-Reference-Cited by-同舟云学术

Cardiac Sodium Channel Na v 1.5 Is Regulated by a Multiprotein Complex Composed of Syntrophins and Dystrophin

Published:2006-08-18 Issue:4 Volume:99 Page:407-414
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Gavillet Bruno¹,Rougier Jean-Sébastien¹,Domenighetti Andrea A.¹,Behar Romina¹,Boixel Christophe¹,Ruchat Patrick¹,Lehr Hans-Anton¹,Pedrazzini Thierry¹,Abriel Hugues¹

Affiliation:

1. From the Department of Pharmacology and Toxicology (B.G., J.-S.R., R.B., C.B., H.A.), University of Lausanne, Switzerland; Service of Cardiology (H.A.), CHUV, Lausanne, Switzerland; Department for Cardiovascular Surgery (P.R.), CHUV, Lausanne, Switzerland; Institute of Pathology (H.-A.L.), CHUV, Lausanne, Switzerland; Deparment of Medicine (A.A.D., T.P.), CHUV, Lausanne, Switzerland.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference40 articles.

1. Genetic control of sodium channel function

2. Regulation of the Voltage-Gated Cardiac Sodium Channel Nav1.5 by Interacting Proteins

3. Nav1.5 E1053K mutation causing Brugada syndrome blocks binding to ankyrin-G and expression of Nav1.5 on the surface of cardiomyocytes

4. Interaction of Muscle and Brain Sodium Channels with Multiple Members of the Syntrophin Family of Dystrophin-Associated Proteins

5. Syntrophin γ2 Regulates SCN5A Gating by a PDZ Domain-mediated Interaction

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