Cardiac Sodium Channel Na v 1.5 Is Regulated by a Multiprotein Complex Composed of Syntrophins and Dystrophin-Reference-Cited by-同舟云学术

Cardiac Sodium Channel Na v 1.5 Is Regulated by a Multiprotein Complex Composed of Syntrophins and Dystrophin

Published:2006-08-18 Issue:4 Volume:99 Page:407-414
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Gavillet Bruno¹,Rougier Jean-Sébastien¹,Domenighetti Andrea A.¹,Behar Romina¹,Boixel Christophe¹,Ruchat Patrick¹,Lehr Hans-Anton¹,Pedrazzini Thierry¹,Abriel Hugues¹

Affiliation:

1. From the Department of Pharmacology and Toxicology (B.G., J.-S.R., R.B., C.B., H.A.), University of Lausanne, Switzerland; Service of Cardiology (H.A.), CHUV, Lausanne, Switzerland; Department for Cardiovascular Surgery (P.R.), CHUV, Lausanne, Switzerland; Institute of Pathology (H.-A.L.), CHUV, Lausanne, Switzerland; Deparment of Medicine (A.A.D., T.P.), CHUV, Lausanne, Switzerland.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference40 articles.

1. Genetic control of sodium channel function

2. Regulation of the Voltage-Gated Cardiac Sodium Channel Nav1.5 by Interacting Proteins

3. Nav1.5 E1053K mutation causing Brugada syndrome blocks binding to ankyrin-G and expression of Nav1.5 on the surface of cardiomyocytes

4. Interaction of Muscle and Brain Sodium Channels with Multiple Members of the Syntrophin Family of Dystrophin-Associated Proteins

5. Syntrophin γ2 Regulates SCN5A Gating by a PDZ Domain-mediated Interaction

Cited by 206 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Empagliflozin treatment rescues abnormally reduced Na⁺ currents in ventricular cardiomyocytes from dystrophin-deficient mdx mice;American Journal of Physiology-Heart and Circulatory Physiology;2024-02-01

2. The Kir2.1-NaV1.5 Channelosome and its role in Arrhythmias in Inheritable Cardiac Diseases;Heart Rhythm;2024-01

3. Efficacy of exon-skipping therapy for DMD cardiomyopathy with mutations in actin binding domain 1;Molecular Therapy - Nucleic Acids;2023-12

4. Calcium handling dysfunction and cardiac damage following acute ventricular preload challenge in the dystrophin-deficient mouse heart;American Journal of Physiology-Heart and Circulatory Physiology;2023-11-01

5. Maturation of iPSC-derived cardiomyocytes in a heart-on-a-chip device enables modeling of dilated cardiomyopathy caused by R222Q-SCN5A mutation;Biomaterials;2023-10