A G/C Element Mediates Repression of the SM22α Promoter Within Phenotypically Modulated Smooth Muscle Cells in Experimental Atherosclerosis
Author:
Affiliation:
1. From the Department of Molecular Physiology and Biological Physics, University of Virginia, Charlottesville.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Reference30 articles.
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5. Molecular mechanisms of decreased smooth muscle differentiation marker expression after vascular injury
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