Role of Nitric Oxide in Ca2+Sensitivity of the Slowly Activating Delayed Rectifier K+Current in Cardiac Myocytes-Reference-Cited by-同舟云学术

Role of Nitric Oxide in Ca2+Sensitivity of the Slowly Activating Delayed Rectifier K+Current in Cardiac Myocytes

Published:2005-01-07 Issue:1 Volume:96 Page:64-72
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Bai Chang-Xi¹,Namekata Iyuki¹,Kurokawa Junko¹,Tanaka Hikaru¹,Shigenobu Koki¹,Furukawa Tetsushi¹

Affiliation:

1. From the Department of Bio-informational Pharmacology (C.-X.B., J.K., T.F.), Medical Research Institute, Tokyo Medical and Dental University, Tokyo; and the Department of Pharmacology (I.N., H.T., K.S.), Toho University School of Pharmaceutical Sciences, Funabashi city, Chiba, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference28 articles.

1. Calmodulin kinase determines calcium-dependent facilitation of L-type calcium channels

2. Functional Stoichiometry and Local Enrichment of Calmodulin Interacting with Ca 2+ Channels

3. Critical Determinants of Ca2+-Dependent Inactivation within an EF-Hand Motif of L-Type Ca2+ Channels

4. Two components of cardiac delayed rectifier K+ current. Differential sensitivity to block by class III antiarrhythmic agents.

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