Major Contribution of the Medial Amygdala to Hypertension in BPH/2J Genetically Hypertensive Mice-Reference-Cited by-同舟云学术

Major Contribution of the Medial Amygdala to Hypertension in BPH/2J Genetically Hypertensive Mice

Published:2014-04 Issue:4 Volume:63 Page:811-818
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Jackson Kristy L.¹,Palma-Rigo Kesia¹,Nguyen-Huu Thu-Phuc¹,Davern Pamela J.¹,Head Geoffrey A.¹

Affiliation:

1. From the Neuropharmacology Laboratory, Baker IDI Heart and Diabetes Research Institute, Melbourne, Victoria, Australia (K.L.J., K.P.-R., T.-P.N.-H., P.J.D., G.A.H.); and Department of Pharmacology, Monash University, Clayton, Victoria, Australia (K.L.J., G.A.H.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference39 articles.

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2. Global identification of the genes and pathways differentially expressed in hypothalamus in early and established neurogenic hypertension

3. Renin–angiotensin and sympathetic nervous system contribution to high blood pressure in Schlager mice

4. Role of the Sympathetic Nervous System in Schlager Genetically Hypertensive Mice

5. Contribution of the sympathetic nervous system to vascular resistance in conscious young and adult spontaneously hypertensive rats.

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