Angiotensin II Promotes K V 7.4 Channels Degradation Through Reduced Interaction With HSP90 (Heat Shock Protein 90)-Reference-Cited by-同舟云学术

Angiotensin II Promotes K V 7.4 Channels Degradation Through Reduced Interaction With HSP90 (Heat Shock Protein 90)

Published:2018-06 Issue:6 Volume:71 Page:1091-1100
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Barrese Vincenzo¹,Stott Jennifer B.¹,Figueiredo Hericka B.¹,Aubdool Aisah A.²,Hobbs Adrian J.²,Jepps Thomas A.³,McNeish Alister J.⁴,Greenwood Iain A.¹

Affiliation:

1. From the Vascular Research Centre, Institute of Molecular and Clinical Sciences, St George’s, University of London, United Kingdom (V.B., J.B.S., H.B.F., I.A.G.)

2. William Harvey Research Institute, Barts and The London School of Medicine, Queen Mary, University of London, United Kingdom (A.A.A., A.J.H.)

3. Department of Biomedical Sciences, University of Copenhagen, Denmark (T.A.J.)

4. and Reading School of Pharmacy, University of Reading, United Kingdom (A.J.M.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference39 articles.

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5. Vascular KCNQ Potassium Channels as Novel Targets for the Control of Mesenteric Artery Constriction by Vasopressin, Based on Studies in Single Cells, Pressurized Arteries, and in Vivo Measurements of Mesenteric Vascular Resistance

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