Reactive Oxygen Species, NADPH Oxidases, and Hypertension
Author:
Affiliation:
1. From the Division of Cardiology, Department of Medicine, Emory University School of Medicine, Atlanta, Ga.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Internal Medicine
Link
https://www.ahajournals.org/doi/pdf/10.1161/HYPERTENSIONAHA.109.142422
Reference56 articles.
1. NAD(P)H Oxidase Inhibition Attenuates Neuronal Chronotropic Actions of Angiotensin II
2. Oxidative Stress Mediates the Stimulation of Sympathetic Nerve Activity in the Phenol Renal Injury Model of Hypertension
3. Sympathoexcitation by Oxidative Stress in the Brain Mediates Arterial Pressure Elevation in Salt-Sensitive Hypertension
4. Induction of Hypertension and Peripheral Inflammation by Reduction of Extracellular Superoxide Dismutase in the Central Nervous System
5. Genetic Silencing of Nox2 and Nox4 Reveals Differential Roles of These NADPH Oxidase Homologues in the Vasopressor and Dipsogenic Effects of Brain Angiotensin II
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