β-Arrestin2 Improves Post–Myocardial Infarction Heart Failure via Sarco(endo)plasmic Reticulum Ca 2+ -ATPase–Dependent Positive Inotropy in Cardiomyocytes-Reference-Cited by-同舟云学术

β-Arrestin2 Improves Post–Myocardial Infarction Heart Failure via Sarco(endo)plasmic Reticulum Ca 2+ -ATPase–Dependent Positive Inotropy in Cardiomyocytes

Published:2017-11 Issue:5 Volume:70 Page:972-981
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

McCrink Katie A.¹,Maning Jennifer¹,Vu Angela¹,Jafferjee Malika¹,Marrero Christine¹,Brill Ava¹,Bathgate-Siryk Ashley¹,Dabul Samalia¹,Koch Walter J.¹,Lymperopoulos Anastasios¹

Affiliation:

1. From the Laboratory for the Study of Neurohormonal Control of the Circulation, Department of Pharmaceutical Sciences, Nova Southeastern University College of Pharmacy, Fort Lauderdale, FL (K.A.M., J.M., A.V., M.J., C.M., A.B., A.B.-S., S.D., A.L.); and Department of Pharmacology, Center for Translational Medicine, Lewis Katz School of Medicine, Temple University, Philadelphia, PA (W.J.K.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference33 articles.

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3. Emerging Functional Divergence of β-Arrestin Isoforms in GPCR Function

4. Arrestins in the Cardiovascular System

5. Crystal Structure of Arrestin-3 Reveals the Basis of the Difference in Receptor Binding Between Two Non-visual Subtypes

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