Intrarenal Angiotensin III Infusion Induces Natriuresis and Angiotensin Type 2 Receptor Translocation in Wistar-Kyoto but not in Spontaneously Hypertensive Rats
Author:
Affiliation:
1. From the Division of Endocrinology and Metabolism, Department of Internal Medicine, University of Virginia Health System, Charlottesville.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Internal Medicine
Reference33 articles.
1. Angiotensin II causes hypertension and cardiac hypertrophy through its receptors in the kidney
2. Distinct roles for the kidney and systemic tissues in blood pressure regulation by the renin-angiotensin system
3. Renal Angiotensin Type 2 Receptors Mediate Natriuresis Via Angiotensin III in the Angiotensin II Type 1 Receptor–Blocked Rat
4. Conversion of Renal Angiotensin II to Angiotensin III Is Critical for AT 2 Receptor–Mediated Natriuresis In Rats
5. Intrarenal Aminopeptidase N Inhibition Augments Natriuretic Responses to Angiotensin III in Angiotensin Type 1 Receptor–Blocked Rats
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