Proinflammatory Effects of Cardiotonic Steroids Mediated by NKA α-1 (Na+/K+-ATPase α-1)/Src Complex in Renal Epithelial Cells and Immune Cells-Reference-Cited by-同舟云学术

Proinflammatory Effects of Cardiotonic Steroids Mediated by NKA α-1 (Na+/K+-ATPase α-1)/Src Complex in Renal Epithelial Cells and Immune Cells

Published:2019-07 Issue:1 Volume:74 Page:73-82
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Khalaf Fatimah K.¹,Dube Prabhatchandra¹,Kleinhenz Andrew L.¹,Malhotra Deepak¹,Gohara Amira²,Drummond Christopher A.¹,Tian Jiang¹,Haller Steven T.¹,Xie Zijian³,Kennedy David J.¹

Affiliation:

1. From the Department of Medicine (F.K.K., P.D., A.L.K., D.M., C.A.D., J.T., S.T.H., D.J.K.) University of Toledo College of Medicine and Life Sciences, OH

2. Department of Pathology (A.G.) University of Toledo College of Medicine and Life Sciences, OH

3. Marshall Institute for Interdisciplinary Research, Marshall University, Huntington, WV (Z.X.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference46 articles.

1. Endogenous Cardiotonic Steroids: Physiology, Pharmacology, and Novel Therapeutic Targets

2. Cardiotonic Steroids and the Sodium Trade Balance: New Insights into Trade-Off Mechanisms Mediated by the Na+/K+-ATPase

3. Marinobufagenin Stimulates Fibroblast Collagen Production and Causes Fibrosis in Experimental Uremic Cardiomyopathy

4. The cardiotonic steroid hormone marinobufagenin induces renal fibrosis: implication of epithelial-to-mesenchymal transition

5. Passive Immunization Against Marinobufagenin Attenuates Renal Fibrosis and Improves Renal Function in Experimental Renal Disease

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