Neuroprotection of Ischemic Postconditioning by Downregulating the Postsynaptic Signaling Mediated by Kainate Receptors-Reference-Cited by-同舟云学术

Neuroprotection of Ischemic Postconditioning by Downregulating the Postsynaptic Signaling Mediated by Kainate Receptors

Published:2013-07 Issue:7 Volume:44 Page:2031-2035
ISSN:0039-2499
Container-title:Stroke
language:en
Short-container-title:Stroke

Author:

Liu Jing¹,Xu Qin¹,Wang Hui¹,Wang Ran¹,Hou Xiao-Yu¹

Affiliation:

1. From the Department of Pharmacology, Nanjing General Hospital of Nanjing Military Command, Jiangsu, China (J.L., X.-Y.H.); and Jiangsu Key Laboratory of Brain Disease Bioinformation (J.L., Q.X., H.W., R.W., X.-Y.H.), Research Center for Biochemistry and Molecular Biology (J.L., R.W., X.-Y.H.), Xuzhou Medical College, Jiangsu, China.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Advanced and Specialized Nursing,Cardiology and Cardiovascular Medicine,Neurology (clinical)

Reference10 articles.

1. From Rapid to Delayed and Remote Postconditioning: The Evolving Concept of Ischemic Postconditioning in Brain Ischemia

2. Emerging mechanisms of disrupted cellular signaling in brain ischemia

3. Neuroprotection against ischaemic brain injury by a GluR6-9c peptide containing the TAT protein transduction sequence

4. Overexpression of the PDZ1 domain of PSD-95 diminishes ischemic brain injury via inhibition of the GluR6·PSD-95·MLK3 pathway

5. In Vivo and In Vitro Characterization of a Novel Neuroprotective Strategy for Stroke: Ischemic Postconditioning

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