Ischemic Postconditioning Protects Against Global Cerebral Ischemia/Reperfusion-Induced Injury in Rats-Reference-Cited by-同舟云学术

Ischemic Postconditioning Protects Against Global Cerebral Ischemia/Reperfusion-Induced Injury in Rats

Published:2008-03 Issue:3 Volume:39 Page:983-990
ISSN:0039-2499
Container-title:Stroke
language:en
Short-container-title:Stroke

Author:

Wang Jing-ye¹,Shen Jia¹,Gao Qin¹,Ye Zhi-guo¹,Yang Shui-you¹,Liang Hua-wei¹,Bruce Iain C.¹,Luo Ben-yan¹,Xia Qiang¹

Affiliation:

1. From the Department of Neurology, First Affiliated Hospital (J.-y.W., B.-y.L.); the Department of Physiology (J.-y.W., J.S., Q.G., Z.-g.Y., H.-w.L., I.C.B., Q.X.); and the Morphology Center (S.-y.Y.), Zhejiang University School of Medicine, Hangzhou, China.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Advanced and Specialized Nursing,Cardiology and Cardiovascular Medicine,Neurology (clinical)

Reference35 articles.

1. Neuroprotective effects of preconditioning ischaemia on ischaemic brain injury through inhibition of mixed-lineage kinase 3 via NMDA receptor-mediated Akt1 activation

2. Dynamics of intracellular calcium and free radical production during ischemia in pyramidal neurons

3. Increased intracellular Ca2+ concentration in the hippocampal CA1 area during global ischemia and reperfusion in the rat: a possible cause of delayed neuronal death

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