Delayed Treatment With Minocycline Ameliorates Neurologic Impairment Through Activated Microglia Expressing a High-Mobility Group Box1–Inhibiting Mechanism-Reference-Cited by-同舟云学术

Delayed Treatment With Minocycline Ameliorates Neurologic Impairment Through Activated Microglia Expressing a High-Mobility Group Box1–Inhibiting Mechanism

Published:2008-03 Issue:3 Volume:39 Page:951-958
ISSN:0039-2499
Container-title:Stroke
language:en
Short-container-title:Stroke

Author:

Hayakawa Kazuhide¹,Mishima Kenichi¹,Nozako Masanori¹,Hazekawa Mai¹,Mishima Shohei¹,Fujioka Masayuki¹,Orito Kensuke¹,Egashira Nobuaki¹,Iwasaki Katsunori¹,Fujiwara Michihiro¹

Affiliation:

1. From the Department of Neuropharmacology (K.H., K.M., M.N., M.H., S.M., M. Fujioka, N.E., K.I., M.F.), Faculty of Pharmaceutical Sciences, and the Advanced Materials Institute (K.I., M. Fujwara), Fukuoka University, Fukuoka; and the Department of Veterinary Pharmacology (K.O.), School of Veterinary Medicine, Azabu University, Kanagawa, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Advanced and Specialised Nursing,Cardiology and Cardiovascular Medicine,Clinical Neurology

Reference37 articles.

1. Blockade of Microglial Activation Is Neuroprotective in the 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine Mouse Model of Parkinson Disease

2. Tetracyclines inhibit microglial activation and are neuroprotective in global brain ischemia

3. A tetracycline derivative, minocycline, reduces inflammation and protects against focal cerebral ischemia with a wide therapeutic window

4. Minocycline Provides Neuroprotection Against N-Methyl-d-aspartate Neurotoxicity by Inhibiting Microglia

5. Minocycline markedly protects the neonatal brain against hypoxic-ischemic injury

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