Glycoprotein Ibα–Mediated Platelet Adhesion and Aggregation to Immobilized Thrombin Under Conditions of Flow-Reference-Cited by-同舟云学术

Glycoprotein Ibα–Mediated Platelet Adhesion and Aggregation to Immobilized Thrombin Under Conditions of Flow

Published:2006-03 Issue:3 Volume:26 Page:670-675
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Weeterings Cees¹,Adelmeijer Jelle¹,Myles Timothy¹,de Groot Philip G.¹,Lisman Ton¹

Affiliation:

1. From the Thrombosis and Haemostasis Laboratory (C.W., J.A., P.G.d.G., T.L.), Department of Haematology, University Medical Centre Utrecht, the Netherlands; the Institute of Biomembranes (C.W., P.G.d.G., T.L.), Utrecht University, Utrecht, the Netherlands; and the Division of Hematology (T.M.), Department of Medicine, Stanford University School of Medicine, Stanford, Calif.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference27 articles.

1. Binding of Various Thrombin Fractions to Fibrin and the Influence of AT-III on Their Adsorption

2. Clot-bound thrombin is protected from inhibition by heparin-antithrombin III but is susceptible to inactivation by antithrombin III-independent inhibitors.

3. Exosites 1 and 2 Are Essential for Protection of Fibrin-bound Thrombin from Heparin-catalyzed Inhibition by Antithrombin and Heparin Cofactor II

4. Effect of Heparin on the Activation of Factor XI by Fibrin-bound Thrombin

5. Protease-activated receptors 1 and 4 mediate activation of human platelets by thrombin

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