Endothelial Dysfunction and Elevated Blood Pressure in Mas Gene-Deleted Mice-Reference-Cited by-同舟云学术

Endothelial Dysfunction and Elevated Blood Pressure in Mas Gene-Deleted Mice

Published:2008-02 Issue:2 Volume:51 Page:574-580
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Xu Ping¹,Costa-Goncalves Andrey C.¹,Todiras Mihail¹,Rabelo Luiza A.¹,Sampaio Walkyria O.¹,Moura Marina M.¹,Sousa Santos Sergio¹,Luft Friedrich C.¹,Bader Michael¹,Gross Volkmar¹,Alenina Natalia¹,Santos Robson A.S.¹

Affiliation:

1. From the Max-Delbrück-Center for Molecular Medicine (P.X., A.C.C-G., M.T., F.C.L., M.B., V.G., N.A.), Berlin, Germany; the Department of Physiology and Biophysics (A.C.C-G., L.A.R., W.O.S., M.M.M., S.S.S., R.A.S.S.), Federal University of Minas Gerais, Belo Horizonte, Minas Gerais, Brazil; Setor de Fisiologia e Farmacologia (L.A.R.), Instituto de Ciências Biológicas e da Saúde (ICBS), Universidade Federal de Alagoas, Maceió, Brazil; and the Medical Faculty of the Charité (F.C.L.), Franz...

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference40 articles.

1. The mas oncogene encodes an angiotensin receptor

2. Angiotensin-(1–7) is an endogenous ligand for the G protein-coupled receptor Mas

3. G-Protein–Coupled Receptor Mas Is a Physiological Antagonist of the Angiotensin II Type 1 Receptor

4. Angiotensin-(1–7) prevents development of severe hypertension and end-organ damage in spontaneously hypertensive rats treated with l-NAME

5. Chronic angiotensin-(1–7) prevents cardiac fibrosis in DOCA-salt model of hypertension

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