Small Heat Shock Proteins and Protection Against Ischemic Injury in Cardiac Myocytes
Author:
Affiliation:
1. From the Department of Medicine, Divisions of Endocrinology and Metabolism (J.L.M., R.M., W.H.D.) and Cardiology (L.L.B.) and Department of Pharmacology (R.H.-D., L.L.B.), University of California, San Diego.
Abstract
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine
Reference34 articles.
1. Expression of inducible stress protein 70 in rat heart myogenic cells confers protection against simulated ischemia-induced injury.
2. Overexpression of the rat inducible 70-kD heat stress protein in a transgenic mouse increases the resistance of the heart to ischemic injury.
3. Transgenic mice expressing the human heat shock protein 70 have improved post-ischemic myocardial recovery.
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