Targeted Disruption of TGF-β–Smad3 Signaling Leads to Enhanced Neointimal Hyperplasia With Diminished Matrix Deposition in Response to Vascular Injury-Reference-Cited by-同舟云学术

Targeted Disruption of TGF-β–Smad3 Signaling Leads to Enhanced Neointimal Hyperplasia With Diminished Matrix Deposition in Response to Vascular Injury

Published:2005-04-29 Issue:8 Volume:96 Page:904-912
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Kobayashi Kazuki¹,Yokote Koutaro¹,Fujimoto Masaki¹,Yamashita Kimihiro¹,Sakamoto Akemi¹,Kitahara Masaki¹,Kawamura Harukiyo¹,Maezawa Yoshiro¹,Asaumi Sunao¹,Tokuhisa Takeshi¹,Mori Seijiro¹,Saito Yasushi¹

Affiliation:

1. From the Department of Clinical Cell Biology (K.K., K.Y., M.F., H.K., Y.M., S.A., S.M., Y.S.), Chiba University Graduate School of Medicine; Division of Endocrinology and Metabolism (K.Y., Y.S.), Department of Internal Medicine, Chiba University Hospital; Department of Developmental Genetics (K.Y., A.S., T.T.), Chiba University Graduate School of Medicine, Chiba, Japan; and Shiraoka Research Station of Biological Science (M.K.), Nissan Chemical Industries, Ltd, Saitama, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference51 articles.

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3. Expression of transforming growth factor-beta 1 is increased in human vascular restenosis lesions.

4. Control of junB and extracellular matrix protein expression by transforming growth factor-beta 1 is independent of simian virus 40 T antigen-sensitive growth-sensitive growth-inhibitory events.

5. Transforming growth factor-beta stimulates the expression of fibronectin and collagen and their incorporation into the extracellular matrix.

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