Nuclear Targeting of Akt Enhances Ventricular Function and Myocyte Contractility-Reference-Cited by-同舟云学术

Nuclear Targeting of Akt Enhances Ventricular Function and Myocyte Contractility

Published:2005-12-09 Issue:12 Volume:97 Page:1332-1341
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Rota Marcello¹,Boni Alessandro¹,Urbanek Konrad¹,Padin-Iruegas Maria Elena¹,Kajstura Tymoteusz J.¹,Fiore Giuseppe¹,Kubo Hajime¹,Sonnenblick Edmund H.¹,Musso Ezio¹,Houser Steve R.¹,Leri Annarosa¹,Sussman Mark A.¹,Anversa Piero¹

Affiliation:

1. From the Cardiovascular Research Institute (M.R., A.B., K.U., M.E.P.-I., T.J.K., G.F., E.H.S., E.M., A.L., P.A.), Department of Medicine, New York Medical College, Valhalla; Cardiovascular Research Center (H.K., S.R.H.), Temple University, Philadelphia, Pa; and San Diego State University Heart Institute (M.A.S.), Calif; and Tardini-Vitali-Mazza-Olivetti Stem Cell Center (E.M.), Parma, Italy.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference39 articles.

1. A kt ion in the Nucleus

2. Heart Failure

3. The Akt-Glycogen Synthase Kinase 3β Pathway Regulates Transcription of Atrial Natriuretic Factor Induced by β-Adrenergic Receptor Stimulation in Cardiac Myocytes

4. The conserved phosphoinositide 3-kinase pathway determines heart size in mice

5. Akt/Protein Kinase B Promotes Organ Growth in Transgenic Mice

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