Renovascular Hypertension in Mice With Brain-Selective Overexpression of AT 1a Receptors Is Buffered by Increased Nitric Oxide Production in the Periphery-Reference-Cited by-同舟云学术

Renovascular Hypertension in Mice With Brain-Selective Overexpression of AT 1a Receptors Is Buffered by Increased Nitric Oxide Production in the Periphery

Published:2004-09-03 Issue:5 Volume:95 Page:523-531
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Lazartigues Eric¹,Lawrence Andrew J.¹,Lamb Fred S.¹,Davisson Robin L.¹

Affiliation:

1. From the Departments of Anatomy & Cell Biology (E.L., R.L.D.) and Pediatrics (F.S.L.), Roy J. and Lucille A. Carver College of Medicine, University of Iowa, Iowa City; Howard Florey Institute (A.J.L.), University of Melbourne, Victoria, Australia.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference36 articles.

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4. Tissue renin-angiotensin systems in renal hypertension.

5. Antisense inhibition of AT1 receptor mRNA and angiotensinogen mRNA in the brain of spontaneously hypertensive rats reduces hypertension of neurogenic origin

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