Bidirectional Ventricular Tachycardia and Fibrillation Elicited in a Knock-In Mouse Model Carrier of a Mutation in the Cardiac Ryanodine Receptor
Author:
Affiliation:
1. From the Molecular Cardiology (M.C., B.C., M.S., M.R., C.N. S.G.P.) IRCCS Fondazione S. Maugeri, Pavia, Italy; the University of Pavia, Pavia (S.G.P.), Italy; and the Pathology Division (M.S., L.V.), IRCCS Fondazione S. Maugeri, Pavia, Italy.
Abstract
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Link
https://www.ahajournals.org/doi/pdf/10.1161/01.RES.0000169067.51055.72
Reference12 articles.
1. Clinical and Molecular Characterization of Patients With Catecholaminergic Polymorphic Ventricular Tachycardia
2. Arrhythmic disorder mapped to chromosome 1q42–q43 causes malignant polymorphic ventricular tachycardia in structurally normal hearts
3. RyR2 mutations linked to ventricular tachycardia and sudden death reduce the threshold for store-overload-induced Ca2+ release (SOICR)
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