Flow-Dependent Dilation Mediated by Endogenous Kinins Requires Angiotensin AT2Receptors-Reference-Cited by-同舟云学术

Flow-Dependent Dilation Mediated by Endogenous Kinins Requires Angiotensin AT2Receptors

Published:2004-06-25 Issue:12 Volume:94 Page:1623-1629
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Bergaya Sonia¹,Hilgers Rob H.P.¹,Meneton Pierre¹,Dong You¹,Bloch-Faure May¹,Inagami Tadashi¹,Alhenc-Gelas François¹,Lévy Bernard I.¹,Boulanger Chantal M.¹

Affiliation:

1. From the Institut National de la Santé et de la Recherche Médicale (INSERM), Unit 541, Hôpital Lariboisière, Paris, France. Present address for R.H.P.H. is the Department of Pharmacology, Cardiovascular Research Institute Maastricht (CARIM), Maastricht, the Netherlands; for P.M., M.B.-F., F.A.-G. is INSERM Unit 367, Paris, France; and for T.I. is the Department of Biochemistry, Vanderbilt University School of Medicine, Nashville, Tenn.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference40 articles.

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3. Expression of Low-Molecular-Weight Kininogen in Mouse Vascular Smooth Muscle Cells.

4. Ramiprilat enhances endothelial autacoid formation by inhibiting breakdown of endothelium-derived bradykinin.

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