Role of nitric oxide in long-term angiotensin II-induced renal vasoconstriction.
Author:
Affiliation:
1. Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson 39216-4505.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Internal Medicine
Reference23 articles.
1. Furchgott RF: Studies on relaxation of rabbit aorta by sodium nitrite: The basis for the proposal that the acid-activatable inhibitory factor from retractor penis is inorganic nitrite and the endothelium-derived relaxing factor is nitric oxide in Vanhoutte PM (ed): Vasodilatation: Vascular Smooth Muscle Peptides Autonomic Nerves and Endothelium. New York Raven Press Publishers 1988 pp 401-414
2. Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide.
3. Characterization of three inhibitors of endothelial nitric oxide synthase in vitro and in vivo
4. Regional and cardiac haemodynamic effects of NG-nitro-l-arginine methyl ester in conscious, Long Evans rats
5. Endothelium-dependent modulation of angiotensin II-induced contraction in blood vessels
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