Nitric Oxide–Dependent Feedback Loop Regulates Transient Receptor Potential Vanilloid 4 (TRPV4) Channel Cooperativity and Endothelial Function in Small Pulmonary Arteries-Reference-Cited by-同舟云学术

Nitric Oxide–Dependent Feedback Loop Regulates Transient Receptor Potential Vanilloid 4 (TRPV4) Channel Cooperativity and Endothelial Function in Small Pulmonary Arteries

Published:2017-12-02 Issue:12 Volume:6 Page:
ISSN:2047-9980
Container-title:Journal of the American Heart Association
language:en
Short-container-title:JAHA

Author:

Marziano Corina¹²,Hong Kwangseok²,Cope Eric L.²,Kotlikoff Michael I.³,Isakson Brant E.¹²,Sonkusare Swapnil K.¹²⁴

Affiliation:

1. Department of Molecular Physiology and Biological Physics, University of Virginia School of Medicine, Charlottesville, VA

2. Robert M. Berne Cardiovascular Research Center, University of Virginia School of Medicine, Charlottesville, VA

3. Department of Biomedical Sciences, College of Veterinary Medicine, Cornell University, Ithaca, NY

4. Department of Pharmacology, University of Virginia School of Medicine, Charlottesville, VA

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference75 articles.

1. Low intravascular pressure activates endothelial cell TRPV4 channels, local Ca2+ events, and IKCa channels, reducing arteriolar tone

2. Functional architecture of inositol 1,4,5-trisphosphate signaling in restricted spaces of myoendothelial projections

3. Unitary TRPV3 channel Ca2+ influx events elicit endothelium-dependent dilation of cerebral parenchymal arterioles

4. Elementary Ca 2+ Signals Through Endothelial TRPV4 Channels Regulate Vascular Function

5. AKAP150-dependent cooperative TRPV4 channel gating is central to endothelium-dependent vasodilation and is disrupted in hypertension

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