Connexin37‐Dependent Mechanisms Selectively Contribute to Modulate Angiotensin II‐Mediated Hypertension-Reference-Cited by-同舟云学术

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Connexin37‐Dependent Mechanisms Selectively Contribute to Modulate Angiotensin II‐Mediated Hypertension

Published:2019-04-16 Issue:8 Volume:8 Page:
ISSN:2047-9980
Container-title:Journal of the American Heart Association
language:en
Short-container-title:JAHA

Author:

Le Gal Loïc¹,Pellegrin Maxime²,Santoro Tania¹,Mazzolai Lucia²,Kurtz Armin³,Meda Paolo⁴,Wagner Charlotte³,Haefliger Jacques‐Antoine¹

Affiliation:

1. Department of Medicine University of Lausanne Switzerland

2. Division of Angiology Heart and Vessel Department Centre Hospitalier Universitaire Vaudois University of Lausanne Switzerland

3. Department of Physiology University of Regensburg Germany

4. Department of Cell Physiology and Metabolism School of Medicine CMU University of Geneva Switzerland

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference47 articles.

1. Connexins: Key Mediators of Endocrine Function

2. Connexins, renin cell displacement and hypertension

3. Connexins and pannexins: from biology towards clinical targets;Meda P;Swiss Med Wkly,2016

4. Gap junction proteins are key drivers of endocrine function

5. Loss of connexin40 is associated with decreased endothelium-dependent relaxations and eNOS levels in the mouse aorta

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4. Function of connexin 43 and RhoA/LIMK2/Cofilin signaling pathway in transient changes of contraction and dilation of human umbilical arterial smooth muscle cells;The International Journal of Biochemistry & Cell Biology;2022-12

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