DNA Damage Response/TP53 Pathway Is Activated and Contributes to the Pathogenesis of Dilated Cardiomyopathy Associated With LMNA (Lamin A/C) Mutations-Reference-Cited by-同舟云学术

DNA Damage Response/TP53 Pathway Is Activated and Contributes to the Pathogenesis of Dilated Cardiomyopathy Associated With LMNA (Lamin A/C) Mutations

Published:2019-03-15 Issue:6 Volume:124 Page:856-873
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circ Res

Author:

Chen Suet Nee¹²,Lombardi Raffaella¹³,Karmouch Jennifer¹⁴,Tsai Ju-Yun¹⁵,Czernuszewicz Grace¹,Taylor Matthew R.G.²,Mestroni Luisa²,Coarfa Cristian⁶,Gurha Priyatansh¹,Marian Ali J.¹

Affiliation:

1. From the Center for Cardiovascular Genetics, Institute of Molecular Medicine, University of Texas Health Sciences Center at Houston (S.N.C., R.L., J.K., J.-Y.T., G.C., P.G., A.J.M.)

2. Section of Cardiology, University of Colorado–Anschutz Medical Campus, Denver (S.N.C., M.R.G.T., L.M.)

3. Division of Cardiology, Department of Advanced Biomedical Science, University of Naples Federico II, Italy (R.L.)

4. MD Anderson Cancer Center, Houston, TX (J.K.)

5. Thermo Fisher Scientific, Taiwan (J.-Y.T.)

6. Department of Cell Biology, Baylor College of Medicine, Houston, TX (C.C.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference59 articles.

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4. Suppression of Activated FOXO Transcription Factors in the Heart Prolongs Survival in a Mouse Model of Laminopathies

5. Molecular insights into the premature aging disease progeria

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