The Kindlin Family of Adapter Proteins
Author:
Affiliation:
1. From the Department of Molecular Cardiology, Joseph J Jacobs Center for Thrombosis and Vascular Biology, Lerner Research Institute, Cleveland Clinic, OH.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Reference15 articles.
1. Kindlins in FERM adhesion
2. Kindlin-2 (Mig-2): a co-activator of β3 integrins
3. Kindlin-2 controls bidirectional signaling of integrins
4. The UNC-112 Gene in Caenorhabditis elegansEncodes a Novel Component of Cell–Matrix Adhesion Structures Required for Integrin Localization in the Muscle Cell Membrane
5. A point mutation in KINDLIN3 ablates activation of three integrin subfamilies in humans
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1. FERM domain–containing proteins are active components of the cell nucleus;Life Science Alliance;2024-01-31
2. Parkin ubiquitination of Kindlin-2 enables mitochondria-associated metastasis suppression;Journal of Biological Chemistry;2023-06
3. Kindlin-3 deficiency leads to impaired erythropoiesis and erythrocyte cytoskeleton;Blood Advances;2023-04-27
4. Understanding cell-extracellular matrix interactions for topology-guided tissue regeneration;BIOCELL;2023
5. Pan-cancer Analysis Predicts Kindlin-associated Global Mechanochemical Perturbation;2022-11-01
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