Augmented Protein Kinase C-α–Induced Myofilament Protein Phosphorylation Contributes to Myofilament Dysfunction in Experimental Congestive Heart Failure-Reference-Cited by-同舟云学术

Augmented Protein Kinase C-α–Induced Myofilament Protein Phosphorylation Contributes to Myofilament Dysfunction in Experimental Congestive Heart Failure

Published:2007-07-20 Issue:2 Volume:101 Page:195-204
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Belin Rashad J.¹,Sumandea Marius P.¹,Allen Edward J.¹,Schoenfelt Kelly¹,Wang Helen¹,Solaro R. John¹,de Tombe Pieter P.¹

Affiliation:

1. From the Department of Physiology and Biophysics, Center for Cardiovascular Research, University of Illinois at Chicago.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference41 articles.

1. European Perspectives

2. Kinases as therapeutic targets for heart failure

3. Integration of Cardiac Myofilament Activity and Regulation with Pathways Signaling Hypertrophy and Failure

4. Molecular and Integrated Biology of Thin Filament Protein Phosphorylation in Heart Muscle

5. Identification of a Functionally Critical Protein Kinase C Phosphorylation Residue of Cardiac Troponin T

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